August 2009

Banish bowel misery forever—in as little as 10 days
Discover the drug-free cure that can wipe away Crohn’s disease, colitis, IBS and more

You don’t set foot in public unless there’s a bathroom close by. Your abdominal pain can be so severe that it doubles you over—and even wakes you up at night. Each time you sit down to enjoy a meal, you wonder if hours of misery will follow.

You’re in the grips of a serious bowel disorder—and it’s taking over your life.

I’ll bet at some point, when you were been sweating, doubled over in pain, or running to the toilet for the umpteenth time, some friend offered the seemingly useless observation that it “must have been something you ate.”

Turns out, there is more wisdom—and truth—to those words than you may have ever imagined. After years of research, I have discovered that today’s most stubborn bowel disorders—diverticulitis/diverticulosis, Crohn’s disease, ulcerative colitis, and irritable bowel syndrome—are all being caused by the same thing.

And it is, indeed, something you ate.

In the next few minutes, I’m going to unlock for you the secret cause of (and cure for) these gut-shredding diseases, and show you the exact method I’ve used to help my patients enjoy permanent relief in a matter of days.

But first you need to understand how mainstream medicine is mis-educating doctors about the real cause of bowel disorders, and how a lot of research—along with a little luck—has led me to the cure.

Stomach pain cured—at 30,000 feet

At some point in your life, you’ve probably been given penicillin to help fight off an infection. But did you know that this world-famous antibiotic was discovered completely by accident? After a long day of bacteria research, Sir Arthur Fleming mistakenly left a petri dish out, and fungus grew on it. It was only later that he realized the bacteria he was studying couldn’t grow around the fungus. Penicillin was born.

I’m telling you this story to illustrate a point. As much as we’d like to believe that all the cures for today’s most serious diseases are discovered in labs, years of diligent research have convinced me that is just isn’t so. Sometimes, cures and breakthroughs are found through pure, dumb luck—simple as that. If you want more proof, wait until you hear how…

I accidentally discovered the hidden cure for diverticulitis/diverticulosis

A friend and I were on our way back to Montana after a short trip to Las Vegas—home to about 90 percent of medical conventions, as far as I can tell. He had been suffering for years from such a bad case of diverticulitis/diverticulosis (diverticulosis is when you get herniations in your bowels, and diverticulitis occurs when they become infected) that he was thinking of letting a surgeon remove part of his colon. In all his years of suffering, the only thing that ever helped him was a two-week course of antibiotics—but you can’t stay on antibiotics forever.

At any rate, on the flight home, my friend was doubled over in agonizing pain. I was struggling to find some way to help him, when I came to a realization so simple and powerful, that I was embarrassed it had never occurred to me before.

Painful bowels are being caused by a food allergy

Why would such a realization hit me out of the blue on a crowded airplane? After all, throughout my career, I (and pretty much every other doctor) had been taught that diverticulitis/diverticulosis is caused by constipation that puts pressure on the large intestine, forcing parts of it to become herniated.

But here I was, 30,000 feet over Idaho, and I was witnessing something very different. You see, Las Vegas was built on more than blackjack and keno—there are buffets, cheap steaks, and free drinks everywhere. If you’ve ever been there, you know that every meal is a feast. If my friend had eaten too much of a food he was allergic to, that would explain the unbearable pain he was experiencing.

It made sense, but I had to test it. When we got home, I put my friend on a course of prednisone after ordering a specific type of allergy test (more on that in a moment). If you’ve ever taken prednisone, you know that it suppresses the allergic response arm of your immune system. So, if my friend’s condition was truly being caused by an allergy, it should provide relief.

Bingo. Prednisone eased his pain dramatically, and when the allergy tests came back, it turned out he was allergic to barley and oysters. He had eaten a large helping of oysters and drank plenty of barley-based beer in Las Vegas. This was why that two-week course of antibiotics had helped him years ago—he couldn’t drink his favorite beer (which was loaded with barley) while he was on the medication.

Turns out, if you have diverticulitis/diverticulosis, allergies cause your immune system to attack and weaken your bowel walls. That’s when the pressure of constipation can easily cause herniation. My mainstream medical education on diverticulitis/diverti­culosis had only been half right—a phenomenon that has occurred more than once.

At any rate, I was able to cure this friend of the incredible pain of diverticulitis/diverticulosis by simply discovering what foods he was allergic to, cutting those foods out of his life, and relieving his constipation. I’ve had patients who suffered terribly for years, and visited every type of specialist you could imagine, who are now living completely pain-free. And here’s the best part…

Hidden allergies are also the cause of Crohn’s disease, colitis and IBS

It may have taken a crisis to help me stumble on the cure for diverticulitis/diverticulosis (I had been brainwashed—I admit it), but I was on to the link between allergies and other inflammatory bowel diseases years ago.

Of course, I didn’t learn this in my formal medical education. A lot of medical training focuses on teaching doctors how to tell bowel disorders apart, because their symptoms can be similar. A strong attack on the small intestine means Crohn’s disease, a less severe but chronic bowel disorder may be IBS, and a strong attack that occurs throughout the colon usually indicates ulcerative colitis.

Doctors are taught that Crohn’s and ulcerative colitis are autoimmune disorders, so many docs can begrudgingly accept that an allergy may be causing the heightened immune response (although most of them will never diagnose the allergy—more on this in a moment). I’ve had a harder time getting my colleagues to accept that IBS is also caused by an allergy, even though I’ve proven it with my patients time and time again.

The reason mainstream medicine has been so slow to pick up on the link between allergies and bowel disorders is both shocking and sad. The truth is…

Most doctors have never been taught how to conduct thorough allergy testing

Oh, they think they know how to conduct allergy testing. You see, when your immune system encounters an allergen, it responds by releasing antibodies to fight it. The IgE class of food-allergen-induced antibodies is the one doctors are most familiar with, so that’s what they test for. IgE antibodies tend to cause hay fever, hives, and other common allergic reactions.

But in the case of autoimmune disorders like Crohn’s disease, ulcerative colitis, IBS, and diverticulitis/diverticulosis, it’s often the IgG class of antibodies that is released—and most doctors never test for it.

You may have been suffering needlessly from bowel disorders for years because you’ve never had comprehensive allergy testing. I see it all the time. Fortunately, I started testing for IgG antibodies years ago, and it’s allowed me to cure patients who had just about given up hope.

I have consistently found that my patients afflicted with chronic bowel disorders are suffering from an undiagnosed food allergy that is forcing their bodies to attack their digestive systems. I help these patients discover their allergy through IgG testing, rid the food or foods from their lives, and they get better fast.

You may wonder why your bowel disorder showed up later in life—after all, in your younger years you probably had an “iron stomach” and could eat anything you wanted without repercussions. The reason for this is quite simple. Our bodies produce several “repair hormones” that fix the damage that occurs when our immune systems attack us.

When we’re younger, we have higher levels of these hormones, and the damage to our bowel walls is quickly repaired. As we age, our hormone levels taper off, and the repairs can’t keep up with the damage. That’s when the uncomfortable—and often painful—symptoms begin. (If you or your doctor wants to know more about these hormones, order my book, The Body Heals, 2nd Edition at www.thebodyheals.com.)

I should tell you that my cure rate for these digestive disorders is not 100 percent. I have some patients who continue to suffer because they refuse to give up a favorite food. It breaks my heart every time.

But for the vast majority of my patients, no food is worth the pain and discomfort of Crohn’s disease, ulcerative colitis, IBS, or diverticulitis/diverticulosis. We identify the food allergen, they swear it off for good, and many of them…

Experience relief in as little as 10 days

Ask your doctor to conduct a comprehensive allergy test, including screening for IgG antibodies. If he doesn’t know how to conduct one, find a doctor who does. That simple test could have you on your way to enjoying the symptom-free, healthy life you deserve.

I should point out, however, that if you are already taking mainstream prescription medications for your bowel disease, you and your doctor may have more work to do. Because these medications suppress your IgG antibodies, they can make testing a bit tricky. Whether mainstream doctors realize it or not, this is how these drugs help control your symptoms. Unfortunately, symptom control approaches always cause side effects.

For this reason, in order for you to become well enough to wean off your medication, I often need to take a trial-and-error approach to determine what you are allergic to. After that, you’ll need to begin adding those eliminated foods back into your diet before testing will show what food is causing your suffering. Unfortunately, this challenge will provoke a relapse, and you will need to briefly restart your medication while we wait for your test results.

For some patients, it takes up to 30 days for symptoms to disappear—but the relief does come. Their days of pills, pain, frustrating visits to specialists, and endless trips to the bathroom are behind them for good. They have their lives back—and it’s time you got your life back, too.

If you or your doctor desires more proof to support what I am saying, order my book, The Body Heals, 2nd Edition, at www.thebodyheals.com.

Conquer osteoporosis with this proven two-step plan

Some of the most confused patients I treat at my clinic in Montana are women suffering from—or trying to prevent—osteoporosis. And I can’t say I blame them.

Starting around middle age, most women are bombarded with information on this disfiguring, bone-wasting disease, and much of the advice out there is confusing, contradictory, half-true, or just plain wrong.

You’re told to get more calcium—but not too much. Maybe some doctor has informed you that your estrogen levels are to blame, or that your osteoporosis is the result of a decrease in the amount of oxygen your body uses (called VO2 max).

It’s no wonder so many women give up trying to cure their osteoporosis. If this sounds like you, I have some bad news and some good news. The bad news is that you’ve probably never been told what’s most likely causing your osteoporosis—after all, most doctors don’t know. The good news is that, once I reveal it to you, you’ll understand that sometimes…

You can cure osteoporosis in just two steps

I’m confident that this simple plan can change your life for the better—just as it has for countless patients of mine. But first you need to understand what’s most likely causing your bones to degenerate, and how simple it can be to reverse the tide.

The REAL cause of most cases of osteoporosis

Let’s get the estrogen myth out of the way first. If low estrogen levels were really the main cause of osteoporosis, men (instead of women) would be disproportionately affected. After all, they make much less estrogen than women.

While men can and do develop osteoporosis, data from the U.S. Centers for Disease Control and Prevention show that osteoporosis is four times as likely to affect women as men.

Hormones play a role—and I’ll give you all the details in a moment—but the notion that estrogen is some sort of silver bullet is just plain wrong.

The idea that calcium or oxygen deprivation is causing osteoporosis may be convenient, but they miss a much larger issue. When you have osteoporosis, your bone cells aren’t just missing one nutrient—they’re being deprived of just about everything. In fact…

If you have osteoporosis, it could mean your bone cells are literally starving

And that brings us to the true culprit of most cases of standard osteoporosis—low levels of the hormone IGF-1. You’ll recall that I discussed IGF-1 in last month’s article on metabolic syndrome (which you can read in the archives at www.healthrevelations.com) and showed you how IGF-1 deficiency can lead to wasting in your muscles.

Well, the same is true for your bones. When they don’t get enough nutrition, they develop what I call hungry cell syndrome. They begin to waste and, over time, you develop osteoporosis.

And here’s why. Your body has two hormones—IGF-1 and insulin—that are primarily responsible for delivering fuel to your cells. Your levels of IGF-1 fall off as you age, and your body compensates with more insulin. The problem is, although both IGF-1 and insulin basically serve as “fuel nozzles” for delivering nutrition to your cells, most of your body’s cells prefer IGF-1 as the nozzle of choice.

Even worse, much of the insulin your body produces never makes it beyond your liver because, anatomically, it always arrives there first. Each liver cell has 200,000 pure insulin receptors, so your liver ends up hogging much of the insulin you produce and hence becomes the nutritional hog of your body. Making matters worse, this insulin instructs your liver to convert the sugar you are consuming into fat (cholesterol and triglycerides) that accumulates in your arteries, around your midsection, and everywhere else.

So, picture with me what’s happening here. At the same time your levels of IGF-1—the preferred nozzle for delivering fuel to your bone cells—are falling off, the insulin that’s supposed to be picking up the slack for blood sugar control is getting trapped in your liver, building fat that puts even more stress on your skeleton (not to mention your heart!).

The nutrition your bones so desperately need is never delivered.

For most run-of-the-mill osteoporosis, IGF-1 deficiency is the culprit—and it’s not even close to optimal. My research has uncovered that healthy people should have 100 times more IGF-1 in their systems than insulin—and if your bones are degenerating, your ratios are probably nowhere near where they should be.

In fact, if you’re suffering from the pain, hunching and brittle bones that come with osteoporosis, I’d wager that most likely…

Your bones are crying out for more IGF-1

You’d think mainstream doctors would be screaming this fact from the tops of the tallest mountains. But they’re not—and the reasons why may surprise you.

Why your doctor has never been taught about IGF-1 and osteoporosis

Once you’ve read a few issues of my newsletter, you’re probably going to know more about IGF-1 than most doctors do. Considering that low IGF-1 is the culprit behind so many diseases, from diabetes to arthritis to osteoporosis, this is one of the greatest outrages in modern medicine.

There are four main reasons for this (although none of them is a great excuse):

Reason #1: Mainstream medical education inadequately covers IGF-1.

Reason #2: The “thought leaders” that control modern medicine keep changing IGF-1’s name.

Reason #3: The unit of measurement for IGF-1 is confusing.

Reason #4: Most of the labs that measure IGF-1 levels do a poor job.

Let’s start with Reason #1. With all the time today’s doctors spend in medical school and in internships and residencies, you’d think they’d leave the process armed with volumes of knowledge on every hormone the body produces. Well, I’m living proof that this just isn’t so.

Believe it or not, most doctors today are incorrectly taught that insulin is the preferred fuel nozzle for your cells, and they don’t fully understand the role it plays in fat formation. It was only several years ago, when I took a sabbatical from my medical practice after realizing my medical education had inadequately prepared me to truly heal patients, that I discovered the hidden truth about IGF-1.

I buried myself in old, discontinued medical textbooks and discovered that a generation ago, doctors were taught the real truth about IGF-1. The older textbooks informed doctors that IGF-1 was supposed to be responsible for 93 percent of the energy-delivering “fuel nozzle action” for your body and that in a healthy person IGF-1 levels are 100 times higher than insulin.

That knowledge has been buried—or has disappeared completely—from current medical education. I don’t know if this omission is the result of ignorance, conspiracy, or a bit of both, but it greatly disturbs me.

Even worse, IGF-1’s name keeps changing. Over the years it’s been called everything from sulfation factor to nonsuppressible insulin-like activity of the bloodstream, before IGF was settled on. Talk about confusing! Many doctors still don’t know IGF-1 by its current, proper name.

To make matters worse, the unit of measurement for IGF-1 is different from the unit of measurement for insulin, which makes a true, apples-to-apples comparison tricky. Insulin is measured in micro units and micromoles, while IGF-1 is measured in nanograms. That might seem like a minor point, until you realize that it’s keeping your doctor from understanding that your body should have much more IGF-1 than insulin.

Finally, I have found that most doctors use labs that do a poor job of measuring IGF. After some frustrating encounters with these labs, I have settled on Pathology Associates in Spokane, WA. Here’s their contact info, for you and your doctor:

110 West Cliff Dr.
Spokane, WA 99204
(509) 755-8600

Working with Pathology Associates, I have discovered that IGF-1 levels of less than 100 ng/dl indicate “run of the mill” osteoporosis. If your value comes back higher, you and your doctor have more detective work to do, and I will discuss some of this additional workup in future newsletters.

You might have given little or no consideration to which lab your doctor uses. “Let him use one he’s comfortable with,” you’ve probably reasoned. Well, it’s time to take this seriously, because…

If your doctor uses the wrong lab, you might never heal from osteoporosis

It’s that important. And that brings us to the first step of my two-step plan:

Step #1: Get an accurate reading of your IGF-1 levels by using a quality lab. If your levels are below 100 ng/dl, you’re ready for…

Step #2: Work with a doctor who understands IGF-1 to increase your levels of this bone-saving hormone. Once you restore your IGF-1 levels to normal, you’ll have struck a powerful blow against the hungry cell syndrome that is contributing to the bone wasting of osteoporosis.

Of course, as you can probably imagine, the fact that most doctors know little about IGF-1 keeps them from being overly skilled in raising your levels.

Raising your IGF-1 levels requires a strategy that also involves other key hormones. You’ll find a detailed plan in my article “Could this ‘muscle head’ hormone be a miracle cure?” on page 6. I devoted an entire article to raising your levels of these key hormones because, aside from defeating osteoporosis, they also hold the key to curing metabolic syndrome—a stubborn disease I’ve discussed in past newsletter issues.

Simply get your IGF-1 levels checked and work with your doctor, using the plan I’m about to spell out for you. That way, you can actually turn the tide against osteoporosis. I’ve used this plan countless times with patients to help them maintain strong bones well into their advanced years.

You too can stop living in fear of the health risks and disfigurement that can become part of living with osteoporosis. As I promised you at the beginning of this article, the simple solution is just two steps away. If you or your doctor desires more proof about these facts, order my book, The Body Heals, 2nd Edition at www.thebodyheals.com.

Could this “muscle head” hormone be a miracle cure?
Despite their reputation, anabolic steroids hold the key to defeating metabolic syndrome and more

Anabolic steroids have a reputation among the American public that hovers somewhere around cigarettes and asbestos. You can’t hear the words “anabolic steroids” without thinking of muscle-head athletes with violent tempers and no necks, who use dangerous levels of the stuff to improve their performance (all while sending a terrible message to our kids and grandkids).

But what these athletes inject into themselves is quite a bit different from the anabolic steroids I’m going to tell you about. Today’s baseball and football players and other star athletes are pumped full of lab-created steroids that are so unnatural they’re often designed for animals!

The good news is, your body actually makes its own anabolic steroids, and they hold the key to defeating metabolic syndrome (a disease where you are simultaneously overweight, pre-diabetic or diabetic, and are also suffering from elevated blood pressure and cholesterol) and more. In fact, when your body doesn’t produce enough anabolic steroids, it unleashes…

A chain reaction that leaves your health in ruins

You might be surprised to hear this—most of my patients are. Many doctors aren’t taught about the importance of anabolic steroids to overall health, so most folks end up in the dark.

Well, that ends today. I’m going to share with you the exact plan I’ve used to help my patients avoid the health-robbing chain reaction that occurs when their anabolic steroid levels are low. It’s a plan you can use to cure metabolic syndrome and lost lean body components (evidenced by hunching, sagging, and crunching) and start enjoying your best health in years.

But first you need to understand why these unfairly maligned hormones are so critical to your health. Below I will introduce you to why you desperately need anabolic steroids to keep old age at bay. (If you desire more evidence order my book, The Body Heals, 2nd Edition at www.thebodyheals.com.)

Getting to the truth about anabolic steroids

Your body makes two types of steroids—anabolic and catabolic. The easy way to remember the difference is that anabolic steroids (such as testosterone) build up our bodies, while catabolic steroids break them down. As you can imagine, there is a constant tug of war occurring in your body between these sets of steroids.

Anabolic steroids tell our genes when it’s time to begin the process of repairing our bones, muscles, organs, and tissue. So, as the ravages of age—or even disease—cause damage to our bodies, anabolic steroids like testosterone step in to help our genes repair it. By contrast, catabolic steroids break down our bodies’ protein to use as fuel. Your ability to hang on to this protein and keep it from being dismantled by catabolic steroids is critical to your good health.

As I mentioned, there is a daily tug of war occurring between anabolic and catabolic steroids. In the case of the muscle wasting that occurs with metabolic syndrome or the bone degeneration that occurs with osteoporosis, I’m sure you’ll have no trouble believing that the catabolic steroids are winning.

I’m going to explain in a moment how that situation has developed. But before we get to that, you’ll need to understand why…

Anabolic steroids determine if your cells get the fuel they crave

If you read last month’s article on metabolic syndrome or this month’s article on osteoporosis (if you haven’t, go ahead and read it now…I’ll wait), you know that a hormone called IGF-1 is critical to delivering fuel to your body’s cells.

There are two hormones that basically serve as “fuel nozzles” for delivering nutrition to your cells—IGF-1 and insulin. And, as I’ve told you before, IGF-1 is the preferred nozzle for your cells 93 percent of the time. Worse, much of the insulin you produce gets stuck in your liver (there are 200,000 pure insulin receptors on every liver cell), where it directs your liver to turn sugar into fat (cholesterol and triglycerides) that gunks up your arteries.

For this reason, when you’re short on IGF—and insulin tries to pick up the slack for blood sugar control—you end up obese and at risk for heart disease, while the rest of your cells are malnourished. They are malnourished because insufficient fuel nozzles are around beyond your liver.

And here’s the really important part: Anabolic steroids tell your liver how much IGF-1 to make. Without adequate levels of anabolic steroids, you will never make enough IGF-1 to deliver the nutrition your cells need. In addition, before you can release IGF-1, your pituitary gland needs to release Growth hormone (a protein hormone). So with either deficiency, the end result can be the muscle wasting common in metabolic syndrome, as well as the hunching, crunching, and sagging skin you see when you look in the mirror. (Next month, I’ll explain how to make sure you’re producing enough Growth hormone.)

One final insult

So, you may be thinking that as long as you normalize your anabolic steroid levels and produce enough IGF-1, everything will be fine, right? Not so fast.

Remember what I told you earlier—that there is a constant tug of war going on between your anabolic and catabolic steroids. Even if you’re making enough anabolic steroids…and even if those steroids have your liver making enough IGF-1…it may never get released into your bloodstream, where it’s needed.

And it’s all due to the most infamous of the catabolic steroids—cortisol. As I explained, catabolic steroids like cortisol break down your body’s protein, turning it into sugar that can be used for fuel. This was an ancient stress mechanism—in times of duress, cortisol gave our ancestors energy to fight off (or outrun) predators or other threats.

But today’s stresses are mostly psychological, so we never burn off all that excess energy. Our blood sugar stays chronically elevated—even when we’re not eating carbs—and this is a big problem if you are endowed with a robust adrenal secretion of cortisol.

You see, your blood sugar needs to fall to around 70 before your pituitary releases Growth hormone and in turn instructs your liver to release IGF-1. And if you’re dealing with chronic stress—or if you suffer from the overactive adrenals that often accompany metabolic syndrome—you might be making so much cortisol that you blood sugar never gets low enough. In other words…

You might be making IGF-1—but you can’t use it

And wait—it gets worse. You’re likely to become sensitive to carbs because of your chronically elevated blood sugar. And because you don’t have enough IGF-1, your body will try to bring down your blood sugar by producing more insulin. That means more fat (more cholesterol, more triglycerides) and more starving cells.

In some cases, you might even run out of cortisol, which will force your body to make more epinephrine (also known as adrenaline). The end result is that you’ll be anxious and jittery, and may suffer from a rapid heartbeat and paranoid thoughts. (I’ll discuss more on this other extreme of diminished adrenal function in future newsletters.)

But now it’s time for healing

To get your cells the nutrition they need—which will help you defeat metabolic syndrome and other wasting diseases—it’s imperative that you make and release enough IGF-1. In order for that to happen, you need your anabolic steroids to win the tug of war against catabolic steroids like cortisol.

Here are five steps you and your doctor can use to get you on the path to better health.

Step #1: Get your IGF-1 level checked. I use Pathology Associates in Spokane, WA (their contact information is on page 5), because I’ve found most other labs do a poor job of measuring IGF-1. You’ll learn pretty quickly whether you are producing and releasing the IGF-1 your body so desperately needs.

Step #2: Have your levels of cortisol and its metabolites checked using a 24-hour urine test. Many doctors don’t check for cortisol metabolites in the urine. Even fewer understand how to interpret what the metabolites mean. Because so many factors—such as stress or when you last ate—can affect your cortisol levels, you really need to analyze them over a period of time. A simple blood or saliva test is imprecise and just won’t cut it because it relies on inferior technology, ignores the very important steroid metabolites, and only measures an instant in time. For 24-hour urine testing, I use Meridian labs or Rhine labs.

Step #3: Get more exercise. Exercise can boost your levels of revitalizing hormones (especially growth hormone) and IGF-1. Don’t try to exercise yourself to death—just pick a moderate routine you can stick with.

Step #4: Fast between meals. That means no snacking. This should allow your blood sugar to fall enough to stimulate the release of IGF-1.

Step #5: Get a good night’s sleep. This also causes blood sugar to fall and stimulates the release of IGF-1 (since it is your longest fast during the day). Many of my patients with metabolic syndrome have a hard time sleeping, because you can’t sleep while producing cortisol (remember, it spikes your blood sugar levels). If you’re having trouble sleeping, talk to your doctor about some simple, drug-free strategies you can try. Simply preparing for sleep—going to bed early, keeping the room dark, turning off the TV, and not exercising or eating too close to bedtime—can often work wonders.

If your testing reveals any hormone deficiencies, you may need to work with a doctor who is competent in hormone replacement therapy. Insist on bioidentical hormones, which are designed to mimic exactly your body’s natural hormones. This is critical, because steroid hormones communicate with your genes—and you don’t want any message confusion occurring.

But steps 3, 4, and 5 are all simple things you can do on your own. I’ve seen many patients who followed these three steps and started seeing improvements in a matter of weeks. They lose weight, have more energy, and begin to turn the tide on the muscle and bone wasting that can run rampant with metabolic syndrome and osteoporosis.

The plan I’ve just laid out for you is simple and easy to follow—but you need to commit to it. Give it a shot, and see how you’re feeling by the time your next newsletter arrives. If you’re anything like the patients I have successfully treated over the years, you’re about to start feeling a whole lot better.

A quick note for the ladies…

When I tell my female patients that they need to boost their levels of anabolic steroids, some of them get nervous. They picture themselves becoming muscular, growing facial hair, and developing other masculine features.

Not to worry—the main anabolic steroid for women is DHEA, which is made by your adrenals. It’s a sex-neutral hormone, which means it usually doesn’t produce any of these side effects you may worry about. Its role is limited to helping your body regenerate and repair itself, so you can start living healthy and pain-free.

In addition, insist on pharmaceutical DHEA available at compounding pharmacies, and consult with a physician competent in bio-identical hormones, who will counsel you on the wisest route of administration.